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DTSTART:19700308T020000
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DTSTART:19701101T020000
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BEGIN:VEVENT
DTSTAMP:20241017T183512Z
LOCATION:Harbor Ballrooms D-I
DTSTART;TZID=America/Los_Angeles:20241016T142500
DTEND;TZID=America/Los_Angeles:20241016T144500
UID:ncs_NCS 2024_sess118_ccrnt262@linklings.com
SUMMARY:Intravascular or Extravasated Blood More Evil in Subarachnoid Hemo
 rrhage?
DESCRIPTION:Breakout Session\n\nBappaditya Ray (University of Texas Southw
 estern Medical Center)\n\nDevelopment and advancement in neurocritical car
 e have resulted in increased survival of subarachnoid hemorrhage (SAH) pat
 ients, resulting in increased morbidity that has a socioeconomic impact at
  individual and societal levels. The young SAH survivors have long-term co
 gnitive disability. How much of this is from the event itself, how much of
  this is from the lingering effects of the type of treatment received, and
  how much of it is related to the late effects of this event is currently 
 unknown. The proposed talk would summarize the role of extravasated subara
 chnoid blood products in secondary brain injury and discuss how genetic pr
 edisposition (e.g., haptoglobin genotype) may predispose some-more compare
 d to others and whether mere mechanical removal of this extravasated blood
  is enough in decreasing patient morbidity. Additionally, systemic procoag
 ulant and proinflammatory responses will be briefly discussed to highlight
  the role of intravascular blood, the interplay between these two processe
 s in exacerbating secondary brain injury, and how these can be modulated t
 o decrease patient morbidity and improve cognitive outcomes.\n\nTrack: Sci
 ence of Neurocritical Care\n\nFocus Area: Coma, Status Epilepticus, Stroke
 , Subarachnoid Hemorrhage, Traumatic Brain Injury\n\nTarget Audience: Inte
 rmediate, Advanced\n\nSession Chair: Sahar Zafar (Massachusetts General Ho
 spital)
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